Of 10hz

Keywords: pemf devices, pemf, magnetic therapy, earthpulse
Description: 10 Hz Pulsed Electromagnetic Field Therapy (PEMF) database shows extraordinary effects of 10 Hz on mitochondrial integrity, tissue regeneration and pain

In May 2002 we began testing our v.1 EarthPulse™ pulsed electromagnetic field prototype at 9.6 Hz due to a very strong correlation of over 40 years of data (primarily Soviet Bloc countries) suggesting that 10 Hz pulse repetition rate for pulsed electromagnetic field therapy was far more effective on healing various tissues and ailments from pain to optic nerve regeneration.

Read Why 9.6 Hz?  to understand the back-story on how we chose 9.6 Hz over 10 Hz. Over the years it became more and more evident we were correct.

Our system uses biologically friendly frequencies to both tune down the mind for better sleep and tune up the mitochondria so more-ATP accelerates healing.

In 1989 David Hood found chronic (35+ days) 10 Hz stimulation at 10 hours per day increased two critical enzymes of cell respiration by a factor of 3x! Citrate synthase. the “pacer” of the Krebs cycle & cytochrome c oxidase the last enzyme in the electron transport chain. UNBELIEVABLE! This is consistent with improved oxygen use in every one of our satisfied clients.

Testing systemic oxygen consumption / cell respiration in clients since 2005 with a test free-diver’s call the static-breath-hold, that we call the resting-breath-hold (RBH) test; clients find routine improvements of 10-20% in as little as 7 days and up to 100% in 90 days. Cell’s burn oxygen more efficiently, drawing oxygen from blood slower, producing more energy and less waste from every breath. Success rate 96%+. RBH-test gradually improves for years with cumulative nighttime use; up to DOUBLE the original time! That can mean nothing but 2x more efficient rate of oxygen burn & fractions of reactive oxygen species (ROS) creation, subsequently lowering requirement of anti-oxidant protection.

NASA – Goodwin – 10 Hz Pulsed Electromagnetic Field Therapy Quadruples Tissue Regeneration:

In 2003 NASA-Goodwin found (2 years AFTER EarthPulse™ began commercial sales utilizing DC pulsed electromagnetic field @ 9.6 Hz) that 10 Hz pulsed electromagnetic field caused neural tissue regeneration @ 4x baseline with improved 3-D orientation (pg 17); while causing DNA to revert from maturation to developmental! (more than 175 maturation genes and 150 developmental genes pgs. 15-26)

The implications on longevity are staggering! Prior to NASA sanitizing this study, it contained data on 5 and 15 Hz stimulation. What was shown was both 5 and 15 Hz provided 2x neural tissue regeneration and NO genetic effects. A perfect bell-shaped curve around 10 Hz. Then why are pulsed electromagnetic therapy systems using frequencies higher. It makes no sense at all.

Developmental gene signature delays appoptosis / mitosis perhaps indefinitely in the absence of oxidative stress. See Goodwin’s subsequent patents here .

By now you should realize a conspiracy to keep this information from the public. See Gizmo the remarkable 16 y.o. (Sept 2010) Yorkie-Maltese on Sleep on Command™ 9.6Hz since 8 y.o. 10 hours per day (1/2 her life). Plays with food and toys like a puppy with eye’s still clear. Greatest longevity experiment of all time! See PETS  page. Who wants to life forever.

James Tong 10 Hz Pulsed Electromagnetic Field Triples Nerve Synapse Junction Voltage Potential & Doubles Mitochondrial Density:

In 2007 James Tong TRIPLED nerve synapse energy (ATP the fuel that fires all cellular processes) & nearly tripled mitochondrial density at nerve synapse junctions in minutes.

In Tong chart linked above, compare DENSITY of unstimulated mitochondria (white circles) with 10 Hz stimulated (black circles); and in lower chart compare ENERGY at synapse junction unstimulated (white circles) with 10 Hz stimulated (black circles). Change nearly 2x & 3x respectively! EarthPulse™ effects last all day!

Read this study abstract Mouse Study Sheds Light on Hearing Loss in Older Adults  on Science Daily (Nov. 12, 2009) and see that we are right on target with delaying age related mitochondrial deficits by increasing energy output through increased mitochondrial integrity, thus delaying mitosis & ultimately apoptosis.

In the column below you’ll read about the synergistic effect of anti-oxidative supplements (discussed in the Science Daily piece immediately above). Science Digest fails however to back you into the real story…that oxidative stress causes breaks in the mitochondrial DNA (the mtDNA).

Oxidative stress causes damage at the sub-cellular mitochondrial DNA level. OXIDATIVE ELECTRONS ARE CREATED at the MITOCHONDRIAL LEVEL. ….and that kicks in oxidative stress, reduced energy production, mitosis and/or appoptosis (programmed cell death). It is same regardless of tissue type. This is the crux of the Mitochondrial Theory of Aging (formerly known as the Free-Radical Theory of Aging). See MoreATP: the Mitochondrial Theory of Aging in Reverse .

When sufficient mitochondrial DNA strand breaks occur and are left unchecked, subsequently the overall energy output of each individual mitochondrion drops. At the cellular level energy drops sufficiently and the mitosis / appoptosis (cell death) switch is thrown.

The mitochondria never sleep! Mitochondria under stimulation by pulsed electromagnetic fields (PEMF) at this very narrow frequency range synthesize more ATP from the oxygen you breath (purely shown through our RBH testing).

This extra energy is available at night for repair, hormone synthesis, memory consolidation and immune support; and during the day for physical and mental performance enhancement (ergogenic effects). More energy out of every breath you take! Now, relate the study below to just about any degenerative disease not mentioned.

There is significant evidence that the pathogenesis of several neurodegenerative diseases, including Parkinson’s disease, Alzheimer’s disease, Friedreich’s ataxia (FRDA), multiple sclerosis and amyotrophic lateral sclerosis, may involve the generation of reactive oxygen species (ROS) and/or reactive nitrogen species (RNS) associated with mitochondrial dysfunction. The mitochondrial genome may play an essential role in the pathogenesis of these diseases, and evidence for mitochondria being a site of damage in neurodegenerative disorders is based in part on observed decreases in the respiratory chain complex activities in Parkinson’s, Alzheimer’s, and Huntington’s disease. Such defects in respiratory complex activities, possibly associated with oxidant/antioxidant imbalance, are thought to underlie defects in energy metabolism and induce cellular degeneration. The impaired intramitochondrial metabolism with increased free iron levels and a defective mitochondrial respiratory chain, associated with increased free radical generation and oxidative damage, may be considered possible mechanisms that compromise cell viability.

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